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Peculiarities of fibrinous pericarditis

Other causes of dry pericarditis are:

  • infectious diseases — tuberculosis, typhoid, dysentery, cholera;
  • renal failure — pericarditis called "death knell" of the final stage of unsuccessful treatment of uremia, death comes 3 weeks after symptoms;
  • myocardial infarction in severe;
  • malignancy due to metastasis from other organs or germination of lung cancer;
  • rare cause of disease of blood and actinomycosis (a fungal infection).

Tuberculosis in 95% of affected lungs. Mycobacterium leprae enters the pericardium from the damaged lung tissue through the pleura. Another way of disintegrating lymph nodes.

Fungal dry pericarditis is called Candida come from. These pathogens are present in the normal any person. They dramatically aktiviziruyutsya and lead to pathology by reducing the immunity. With the bloodstream to the pericardium. Diagnosis is very difficult, but in the case of detection treatment always successful. Subsequently developed adhesions.

What happens in the heart bag?

Inflammation of the pericardium may be only local. Then it covers the base of the heart. When spread on a wide area there is a General diffuse redness of the whole of the serous surface. Due to deposition of fibrin pericardial sheets have a matte finish (not shiny). Can be covered by fibrinous growths in the form of fibers. Such heart is called "villous".

In the development of dry pericarditis is the main role of the accumulated uric acid and urea. They move from the blood into pericardial fluid, causing irritation, then inflammation fibrinous and exudative in nature. In addition, there are conditions for infection.

In this way it may be pericarditis in patients of dialysis centers. Harmful substances are eliminated, but created a "gateway" for the penetration of germs.

Inflammation of pericardial bags after the heart attack occurs in ¼ of patients. Most often it occurs on the background of the transmural changes when the area of necrosis extended to the entire thickness of the myocardium.

Distinguish 2 types:

  1. early — occurs in the first days of acute myocardial infarction;
  2. late — called syndrome W. Dressler, also fibrinous pericarditis, include two - or unilateral pleural effusion.

The mechanism of pathology believe allergic process in the necrotic tissue of the heart. In the pericardial fluid revealed high levels of eosinophils.

Clinical manifestations

In most cases, in dry pericarditis cardiac symptoms are moderate. In patients on the background of the underlying disease appear:

  • heart pain aching, stabbing or pressing character, they become stronger whenthe movement of the body, tilting the head, cough tremors, and sometimes in swallowing;
  • in children common symptom is a hiccup;
  • the temperature is not decreasing, but increasing.

About the accumulation of fluid within the pericardial cavity says the changing nature of pain: they become similar to angina at rest, increasing intensity, radiating to back and shoulder.